The paper deals with ultrasound effects examined in experiments on 68 male Wistar rats with experimental pneumonia and on animals with pneumonia injected with plasma from rats pre exposed to ultrasound. Ultrasound is shown to inhibit pulmonary inflammation and lipid peroxidation. The role of humoral factors in preventive mechanism of ultrasound action is elucidated.

The paper deals with ultrasound effects examined in experiments on 68 male Wistar rats with experimental pneumonia and on animals with pneumonia injected with plasma from rats pre exposed to ultrasound. Ultrasound is shown to inhibit pulmonary inflammation and lipid peroxidation. The role of humoral factors in preventive mechanism of ultrasound action is elucidated.

When irradiating rats with sources of UV-radiation of different spectral composition a decrease in a relative content of cholesterol is found in the animal skin. Simultaneously an increase in the content of such cholestorol biosynthesis precursors as lathosterol and 7-dehydrocholesterol is observed. The formation of cholesterol photooxidation products under the effect of UV-radiation in vivo is shown. The quantitative ratios of sterols under study depend on spectral characteristics of the applied UV-radiation sources. The cholesterol photooxidation products and antirachitic compounds formed in skin under the effect of UV-radiation are supposed to take part in inhibition of cholesterol biosynthesis.

Hypoxic trainings of rats (maintenance in the test chamber at the "altitude" 4 km above the sea level for 7 hours a day for two weeks) prevent pneumonia-induced activation of peroxidation for lipids of the liver mitochondria. This increases the phosphorylating respiration rate when lipemic serum is used as an oxidation substrate (not succinate). In these experiments the efficiency of oxidative phosphorylation (delta ADP/delta O) when either succinate and glutamate or glutamate and malate have been oxidated corresponded to the values typical of intact animals and was higher when lipemic serum was used. It is supposed that rearrangement of energy reactions of mitochondria is connected with intensification of utilization of lipids and conjugation between their oxidation and phosphorylation. This rearrangement is apparently aimed to prevent the energy deficiency in the organism which arises in patients with pneumonia.